Lithgow Laser Clinic


Known as the 'Non-surgical facelift', this treatment targets many concerns in  the one treatment-collagen loss (wrinkles), telangiectasis (broken capillaries), rosacea, pigmentation (sun damage,freckling & melasma), acne, large open pores, scars as well as lightening and brightening the skin. It also reduces redness and stimulates collagen growth -firming and tightening the skin approximately 3 to 4mm. The collagen takes 12 weeks to fully develop and lasts approximately 2 years. The main benefit is that there is very little and in most cases, no down time after the treatments. Treatment of the whole face takes approximately 20 minutes and the face & neck about 30 minutes. If you are having a microdermabrasion treatment prior to this you need to allow another 20 minutes.

A test patch is required 48 hours prior to the first treatment-this is a legal requirement. Most clients have this done at the initial consultation. A 'one-off' microdermabrasion treatment immediately prior to the first photorejuvenation treatment is highly recommended. Research has shown that this enables better light penetration and a much more effective treatment.

After a series of 3 to 5 initial treatments which are sheduled 3 to 4 weeks apart, only 2 to 4 are required annually to maintain bright and health looking skin. Please see slide show at the bottom of this page for before and after photos.

Photorejuvenation treatments can be substantially further enhanced when used in conjuction with; 
  • Age Intervention Regeneration booster: telemerase enzyme, TGF Beta 1, skin growth factors, antioxidants and peptides. This technology was awarded the Nobel Prize for Telomere and Telomerase research! See before and after photos at bottom of Jan Marini Skin Research page.
  • Retinol Plus: the most aggressive levels of stabilized all-tans-retinol PLUS anti-ageing and collagen boosting peptides, antioxidants, hydrators and skin soothing agents. See before and after photos at bottom of Jan Marini Skin Research page.
  • Bioglycolic Cream:  a hydroxyacid which works as an exfoliant giving skin a smoother, brighter, dewy complexion.
  • C-ESTA & DMAE complex: ascorbital palmitate and Dimethyl amino ethanol, a potent antioxidant and acetylcholine precursor, zinc and pyridoxine. Strengthens blood vessel walls and connective tissue, firming skin, lightening pigment and reducing redness/capillaries.
Consistent and diligent use of physical and chemical sun-protection measures using a high end UVA & UVB sunblock  EVERY DAY cannot be emphasised enough to protect skin from further damage from UV light. This will substantially improve your skin over time and it is accumulative.

Sun damage and ageing skin
Skin ageing is a complex and varied process. Many factors influence how a particular individual's face will age. Biological ageing due to heredity generally does not occur until the age of 70 or later and consists primarily of a loss of tissue as well as receding bone structure
that result in a gaunt, sagging appearance.

It is estimated that as much as 96% of the visible signs of ageing is the result of accumulated sun damage. These include; fine lines, wrinkles, textural changes, sun spots (solar lentigines), elastosis (loss of elasticity), coarse dryness, telangiectasia/poikiloderma/spider veins/broken capillaries (blood vessel damage), skin growths, breakdown of collagen and yellow or grey skin hues due to a decrease in the small blood vessels that provide circulation to the surface of the skin. Collectively, these changes  caused by accummulated sun exposure can be grouped into a category often referred to as 'quality of the skin'.

Accelerated skin ageing that is caused by cumulative sun exposure is a complex reactive process. This process consists of countless changes that involve damaged DNA biochemistry, breakdown of cell membranes and destructive effects on enzymes, proteins and amino acids, and abnormal changes to the immune system that are essential for information processing necessary for health youthful skin.

Free radicals are oxygen molecules that have become highly reactive, usually by exposure to UV light (sunlight). The highly reactive molecules result in damage to the cell membrane. When free radicals attack, it usually culminates in a kind of cascading effect that results in hundreds of thousands of cells being damaged in a split second. Even minimal sun exposure triggers an intense invasion of free radicals due to the release of inflammatory cells called
mast cells that come from the lymph nodes. These inflammatory cells appear because UV damage causes the cells to perceive that a bacterial invasion has taken place. The cells try to detroy the perceived bacterial invaders by releasing reactive oxygen molecules called free radicals. However, because there are no microbes for the free radicals to attack, the free radical molecules begin a destructive process that damages or destroys the cell membrane. Consistant release of free radicals has proven to result in the following long-term damage;  
  • Substantial destruction of collagen and elastin (collagen and elastin are the primary components of the skin's support structure)
  • Loss of muscle tone-skin becomes slack and loses its ability to restore its original shape
  • Lines, wrinkles and dry skin
  • Coarseness and textural changes
  • Skin discolouration (pigmentation/brown spots)
  •  Telangiectasia/poikiloderma/spider veins/broken capillaries (blood vessel damage)
  • Damage to DNA
  • Impairment of the skin's immune function which leads to skin cancer
  • Weakening and destruction of cells that come into contact with free radicals

Please contact Elke for a fact sheet on sundamage and ageing, melasma, rosacea or spider veins.

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